Nevertheless, the gain of function research with coronaviruses has been documented in the peer-reviewed literature.
The lack of new information from the Wuhan Institute, despite its longstanding research activities, is probably the most compelling evidence in support of the lab escape hypothesis.
If the evidence pointed elsewhere, it would be released. The most likely explanation is that the Institute's fingerprints are all over this thing.
The second most compelling evidence is that to date the reservoir species has not been found.
Instead, nothing. In their perfect moment of glory and justification, they prefer you didn't notice their work.
Researchers from WIV collaborated with UNC Chapel Hill in 2015 to do that kind of research on SARS-1-related viruses, and that happened in the US.
That gain of function experiment in mice was also to gain function in mice, it would reduce its ability to infect humans.
And if you took RaTG13 and spliced in the surface protein of SARS-1 so it bound to ACE and then ran it through mice you'd still not wind up with SARS-CoV-2. The original virus is too far distant (only 96% homology) and running it through mice would produce a virus that was poorly adapted to humans. And the spike protein would look more like SARS-CoV-1.
https://thebulletin.org/2021/05/the-origin-of-covid-did-peop...